Anetoderma
| Anetoderma | |
|---|---|
| Synonyms | Macular atrophy |
| Pronounce | N/A |
| Specialty | N/A |
| Symptoms | Skin lesions, atrophy |
| Complications | None typically |
| Onset | Usually in adolescence or early adulthood |
| Duration | Chronic |
| Types | N/A |
| Causes | Unknown, possibly autoimmune or genetic factors |
| Risks | Family history, autoimmune disease |
| Diagnosis | Clinical diagnosis, skin biopsy |
| Differential diagnosis | Pseudoxanthoma elasticum, mid-dermal elastolysis, cutaneous mastocytosis |
| Prevention | N/A |
| Treatment | No definitive treatment, options include topical and systemic therapies |
| Medication | N/A |
| Prognosis | Benign, cosmetic concern |
| Frequency | Rare |
| Deaths | N/A |
Anetoderma is a localized skin condition with its characteristic button hole type leisons.
Cause[edit]
It is due to abnormal laxity of the skin with herniation or outpouching resulting from abnormal dermal elastic tissue.
Types[edit]
- Primary anetoderma
- Jadassohn–Pellizzari anetoderma
- Schweninger–Buzzi anetoderma
- Secondary anetoderma
- Familial anetoderma
Secondary anetoderma[edit]
Any of the following conditions can cause secondary anetoderma
- Infections such as chickenpox, syphilis
- Tumours such as pilomatrixoma, plasmacytoma, schwannoma, cutaneous B-cell lymphoma
- Infantile haemangioma
- Inflammatory skin conditions such as acne, granuloma annulare, lupus profundus
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