Warfarin resistance

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Warfarin Resistance

Warfarin resistance is a medical condition where the body's response to the anticoagulant drug Warfarin is reduced, leading to a decreased effectiveness of the drug.

Pronunciation

Warfarin resistance: /wɔːrfərɪn rɪˈzɪstəns/

Etymology

The term "Warfarin" is derived from the acronym WARF, for the Wisconsin Alumni Research Foundation + the ending -arin indicating its link with coumarin, the chemical compound it is derived from. "Resistance" comes from the Latin resistere, meaning "to withstand".

Definition

Warfarin resistance can be defined as a decreased susceptibility to the effects of the anticoagulant drug Warfarin. This can be due to genetic factors, dietary habits, or interactions with other drugs.

Symptoms

Symptoms of Warfarin resistance may not be immediately apparent, as they are often similar to the conditions for which Warfarin is prescribed. These can include increased clotting, deep vein thrombosis, and pulmonary embolism.

Causes

Warfarin resistance can be caused by a variety of factors. Genetic mutations in the VKORC1 gene, which is responsible for the production of Vitamin K epoxide reductase, the enzyme Warfarin targets, can lead to resistance. Additionally, dietary habits such as high Vitamin K intake can reduce the effectiveness of Warfarin. Interactions with other drugs can also lead to Warfarin resistance.

Treatment

Treatment for Warfarin resistance often involves adjusting the dosage of Warfarin or switching to a different anticoagulant drug. Genetic testing may be used to determine the cause of the resistance and guide treatment decisions.

Related Terms

External links

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