Optic disc drusen: Difference between revisions
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{{Infobox medical condition | |||
| name = Optic disc drusen | |||
| image = [[File:Optic_disc_drusen_73jw_-_CT_axial_-_001.jpg]] | |||
| caption = CT scan showing optic disc drusen | |||
| field = [[Ophthalmology]] | |||
| synonyms = Drusen of the optic nerve head | |||
| symptoms = [[Visual field]] defects, [[vision loss]] | |||
| complications = [[Non-arteritic anterior ischemic optic neuropathy]] | |||
| onset = Usually detected in [[childhood]] or [[adolescence]] | |||
| duration = [[Chronic (medicine)|Chronic]] | |||
| causes = [[Genetic predisposition]] | |||
| risks = Family history of optic disc drusen | |||
| diagnosis = [[Ophthalmoscopy]], [[Optical coherence tomography]], [[Ultrasound]] | |||
| differential = [[Papilledema]], [[Optic neuritis]] | |||
| treatment = Monitoring, [[Laser therapy]] for complications | |||
| prognosis = Variable, may lead to [[vision loss]] | |||
| frequency = 0.3% to 2% of the population | |||
}} | |||
'''Optic disc drusen''' (ODD) are globules of protein and calcium salts that accumulate in the optic nerve, particularly in the anterior portion. They are generally benign and are often discovered incidentally during routine eye examinations. However, in some cases, they can lead to visual field defects or other complications. | '''Optic disc drusen''' (ODD) are globules of protein and calcium salts that accumulate in the optic nerve, particularly in the anterior portion. They are generally benign and are often discovered incidentally during routine eye examinations. However, in some cases, they can lead to visual field defects or other complications. | ||
==Etiology== | ==Etiology== | ||
The exact cause of [[optic disc drusen]] is unknown. They are thought to result from abnormal metabolism of retinal ganglion cells, which leads to the accumulation of waste products in the optic nerve. Some studies suggest a genetic component, as ODD has been found to occur more frequently in certain families. | The exact cause of [[optic disc drusen]] is unknown. They are thought to result from abnormal metabolism of retinal ganglion cells, which leads to the accumulation of waste products in the optic nerve. Some studies suggest a genetic component, as ODD has been found to occur more frequently in certain families. | ||
==Clinical Presentation== | ==Clinical Presentation== | ||
Patients with optic disc drusen are often asymptomatic. When symptoms do occur, they may include [[visual field defects]], transient visual obscurations, and, rarely, [[visual loss]]. The drusen themselves are typically visible as yellowish refractile bodies on the surface of the optic disc. | Patients with optic disc drusen are often asymptomatic. When symptoms do occur, they may include [[visual field defects]], transient visual obscurations, and, rarely, [[visual loss]]. The drusen themselves are typically visible as yellowish refractile bodies on the surface of the optic disc. | ||
==Diagnosis== | ==Diagnosis== | ||
Diagnosis of optic disc drusen is typically made through ophthalmoscopic examination. Additional diagnostic tests may include [[fluorescein angiography]], optical coherence tomography (OCT), and visual field testing. In some cases, ultrasound may be used to confirm the diagnosis. | Diagnosis of optic disc drusen is typically made through ophthalmoscopic examination. Additional diagnostic tests may include [[fluorescein angiography]], optical coherence tomography (OCT), and visual field testing. In some cases, ultrasound may be used to confirm the diagnosis. | ||
==Treatment and Prognosis== | ==Treatment and Prognosis== | ||
There is currently no cure for optic disc drusen. Treatment is generally aimed at managing any associated symptoms and monitoring for potential complications. Regular eye examinations are recommended for individuals with ODD to monitor for changes in vision or the development of complications such as [[neovascularization]] or [[optic nerve compression]]. | There is currently no cure for optic disc drusen. Treatment is generally aimed at managing any associated symptoms and monitoring for potential complications. Regular eye examinations are recommended for individuals with ODD to monitor for changes in vision or the development of complications such as [[neovascularization]] or [[optic nerve compression]]. | ||
==See Also== | ==See Also== | ||
* [[Optic nerve]] | * [[Optic nerve]] | ||
* [[Visual field test]] | * [[Visual field test]] | ||
* [[Fluorescein angiography]] | * [[Fluorescein angiography]] | ||
* [[Optical coherence tomography]] | * [[Optical coherence tomography]] | ||
==References== | ==References== | ||
{{reflist}} | {{reflist}} | ||
[[Category:Eye diseases]] | [[Category:Eye diseases]] | ||
[[Category:Optic nerve disorders]] | [[Category:Optic nerve disorders]] | ||
{{Eye-disease-stub}} | {{Eye-disease-stub}} | ||
Latest revision as of 22:13, 6 April 2025
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| Optic disc drusen | |
|---|---|
| |
| Synonyms | Drusen of the optic nerve head |
| Pronounce | N/A |
| Specialty | N/A |
| Symptoms | Visual field defects, vision loss |
| Complications | Non-arteritic anterior ischemic optic neuropathy |
| Onset | Usually detected in childhood or adolescence |
| Duration | Chronic |
| Types | N/A |
| Causes | Genetic predisposition |
| Risks | Family history of optic disc drusen |
| Diagnosis | Ophthalmoscopy, Optical coherence tomography, Ultrasound |
| Differential diagnosis | Papilledema, Optic neuritis |
| Prevention | N/A |
| Treatment | Monitoring, Laser therapy for complications |
| Medication | N/A |
| Prognosis | Variable, may lead to vision loss |
| Frequency | 0.3% to 2% of the population |
| Deaths | N/A |
Optic disc drusen (ODD) are globules of protein and calcium salts that accumulate in the optic nerve, particularly in the anterior portion. They are generally benign and are often discovered incidentally during routine eye examinations. However, in some cases, they can lead to visual field defects or other complications.
Etiology[edit]
The exact cause of optic disc drusen is unknown. They are thought to result from abnormal metabolism of retinal ganglion cells, which leads to the accumulation of waste products in the optic nerve. Some studies suggest a genetic component, as ODD has been found to occur more frequently in certain families.
Clinical Presentation[edit]
Patients with optic disc drusen are often asymptomatic. When symptoms do occur, they may include visual field defects, transient visual obscurations, and, rarely, visual loss. The drusen themselves are typically visible as yellowish refractile bodies on the surface of the optic disc.
Diagnosis[edit]
Diagnosis of optic disc drusen is typically made through ophthalmoscopic examination. Additional diagnostic tests may include fluorescein angiography, optical coherence tomography (OCT), and visual field testing. In some cases, ultrasound may be used to confirm the diagnosis.
Treatment and Prognosis[edit]
There is currently no cure for optic disc drusen. Treatment is generally aimed at managing any associated symptoms and monitoring for potential complications. Regular eye examinations are recommended for individuals with ODD to monitor for changes in vision or the development of complications such as neovascularization or optic nerve compression.
See Also[edit]
References[edit]
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