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Summary
DescriptionEritroblastosis fetalis ku.png
English: When an Rh− mother has an Rh+ fetus, fetal erythrocytes are introduced into the mother’s
circulatory system before or during birth, leading to production of anti-Rh IgG antibodies. These antibodies remain in the mother and, if she becomes pregnant with a second Rh+ baby, they can cross the placenta and attach to fetal Rh+ erythrocytes. Complement-mediated hemolysis of fetal erythrocytes results in a lack of sufficient cells for proper oxygenation of the fetus. HDN can be prevented by administering Rho(D) immune globulin during and after each pregnancy with an Rh+ fetus. The immune globulin binds fetal Rh+ RBCs that gain access to the mother’s bloodstream, preventing activation of her primary immune response.
Kurdî: Heke xwîna dayik Rh negatîv, xwîna bav Rh pozîtîv be û xwîna korpele jî wekî mîna xwîna bavê xwe Rh pozîtîv be, nexweşî peyda dibe.Heke xwîna korpele Rh pozîtiv û ya dayikê Rh negativ be, dijepeydakerên li ser rûyê xirokên sor ên korpeleyê sîstema bergiriya dayikê çalak dike. Ji boy rê li ber nexweşiya ji ber neguncaniya Rhê bên girtin, dema ducaniya yekem de dayik bi dijetena Antî-D ve tê derzîkirin.
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{{Information |description ={{en|1=When an Rh− mother has an Rh+ fetus, fetal erythrocytes are introduced into the mother’s circulatory system before or during birth, leading to production of anti-Rh IgG antibodies. These antibodies remain in the mother and, if she becomes pregnant with a second Rh+ baby, they can cross the placenta and attach to fetal Rh+ erythrocytes. Complement-mediated hemolysis of fetal erythrocytes results in a lack of sufficient cells for proper oxygenation of the fe...